Báo cáo y học: Localised pericardial tamponade diagnosed by computed tomography: a case presentation
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Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành y học dành cho các bạn tham khảo đề tài: Localised pericardial tamponade diagnosed by computed tomography: a case presentation...
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Báo cáo y học: "Localised pericardial tamponade diagnosed by computed tomography: a case presentation"Journal of Medical Case Reports BioMed Central Open AccessCase reportLocalised pericardial tamponade diagnosed by computedtomography: a case presentationHunaid A Vohra*1, Hazem Khout1, Deepashree Bapu2 and Qamar Abid1Address: 1Department of Cardiothoracic Surgery, University Hospital of North Staffordshire NHS Trust, Stoke-on-Trent, UK and 2Department ofCardiac Surgery, Harefield Hospital, Royal Brompton & Harefield Hospitals NHS Trust, London, UKEmail: Hunaid A Vohra* - hunaidvohra@yahoo.co.uk; Hazem Khout - hazemkhout@yahoo.com; Deepashree Bapu - deepa@yahoo.com;Qamar Abid - qamar.abid@uhns.nhs.uk* Corresponding authorPublished: 1 December 2007 Received: 1 March 2007 Accepted: 1 December 2007Journal of Medical Case Reports 2007, 1:162 doi:10.1186/1752-1947-1-162This article is available from: http://www.jmedicalcasereports.com/content/1/1/162© 2007 Vohra et al; licensee BioMed Central Ltd.This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0),which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. blood cardioplegia. The patient was cooled to 32°C. TheIntroductionIn a normovolemic patient, low cardiac output after car- left internal mammary artery was anastomosed to thediac surgery may be a result of myocardial ischaemia and/ LAD, reversed long saphenous vein (LSV) grafts were per-or pericardial tamponade. However, without any objec- formed to posterior descending artery and left ventriculartive evidence of ischaemia alongwith no signs of pericar- branch of RCA as well as obtuse marginal and diagonaldial tamponade or regional wall motion abnormality on arteries (CABG times 5). The CPB time was 85 minutestransthoracic echocardiogram (TTE), the diagnosis and the cross-clamp time was 65 minutes. The heart wasremains ambiguous. Computed tomography (CT scan) of weaned off CPB easily without any inotropes. A left pleu-the chest may be helpful to reveal pericardial tamponade. ral and mediastinal drain was inserted. Following closure of the chest, he was transferred to the intensive care unit (ICU), where he made excellent progress initially and wasCase presentationA 73 year old, hypertensive and hypercholestremic gentle- extubated within 12 hours. At 24 hours post-operatively,man, presented to the Emergency Department with acute the blood pressure (BP) was 110/85 mm Hg, the cardiac index (CI) was 3.0 litres/min/m2 and the total amount ofonset of severe retrosternal chest pain. He had no othersignificant co-morbidities. ECG showed ST segment blood in the drains was 1350 mls, with no drainage in thedepression in leads I, AVL, V5 and V6. The troponin I level last 2 hours. Within 2 hours of removing the drains, thewas 4.1 ng/ml. A diagnosis of non-ST elevation myocar- BP dropped to 80/40 mmHg with a CI of 1.8 litres/min/ m2 with no change in the central venous pressure (CVP,dial infarction (NSTEMI) was made. The patient was givenaspirin, clopidogrel and subcutaneous clexane. During 10 mm Hg), whilst the urine output was maintained atthe admission he continued to get chest pain intermit- >0.5 ml/kg/hr. The systemic vascular resistance was 1150 dynes/cm5. No new changes were seen in the ECG.tently, which required intravenous glyceryl trinitrate infu-sion. A coronary angiogram was performed 4 days later,which revealed significant stenosis of the proximal left A TTE was performed by an experienced sonographeranterior descending artery (LAD) and circumflex artery which showed similar left ventricular function as before(Cx) as well as an occluded right coronary artery (RCA) in and no evidence of pericardial collection or tamponade.the mid-vessel. A TTE showed moderately impaired left In view of depressed LV function, 0.05 mcg/kg/min ofventricular ejection fraction (Journal of Medical Case Reports 2007, 1:162 http://www.jmedicalcasereports.com/content/1/1/162and the urine output was 30 mls/hr. Despite a normal namic instability while in the remaining one-third, collec-TTE, a strong suspicion of pericardial tamponade was tions around the right atrium and/or right ventricle are themade. A trans-oesophageal echocardiogram (TOE) was cause [3]. The decision to re-explore the chest should benot available and it was decided to perform a CT scan of based on clinical suspicion derived from signs whichthe ch ...
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Báo cáo y học: "Localised pericardial tamponade diagnosed by computed tomography: a case presentation"Journal of Medical Case Reports BioMed Central Open AccessCase reportLocalised pericardial tamponade diagnosed by computedtomography: a case presentationHunaid A Vohra*1, Hazem Khout1, Deepashree Bapu2 and Qamar Abid1Address: 1Department of Cardiothoracic Surgery, University Hospital of North Staffordshire NHS Trust, Stoke-on-Trent, UK and 2Department ofCardiac Surgery, Harefield Hospital, Royal Brompton & Harefield Hospitals NHS Trust, London, UKEmail: Hunaid A Vohra* - hunaidvohra@yahoo.co.uk; Hazem Khout - hazemkhout@yahoo.com; Deepashree Bapu - deepa@yahoo.com;Qamar Abid - qamar.abid@uhns.nhs.uk* Corresponding authorPublished: 1 December 2007 Received: 1 March 2007 Accepted: 1 December 2007Journal of Medical Case Reports 2007, 1:162 doi:10.1186/1752-1947-1-162This article is available from: http://www.jmedicalcasereports.com/content/1/1/162© 2007 Vohra et al; licensee BioMed Central Ltd.This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0),which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. blood cardioplegia. The patient was cooled to 32°C. TheIntroductionIn a normovolemic patient, low cardiac output after car- left internal mammary artery was anastomosed to thediac surgery may be a result of myocardial ischaemia and/ LAD, reversed long saphenous vein (LSV) grafts were per-or pericardial tamponade. However, without any objec- formed to posterior descending artery and left ventriculartive evidence of ischaemia alongwith no signs of pericar- branch of RCA as well as obtuse marginal and diagonaldial tamponade or regional wall motion abnormality on arteries (CABG times 5). The CPB time was 85 minutestransthoracic echocardiogram (TTE), the diagnosis and the cross-clamp time was 65 minutes. The heart wasremains ambiguous. Computed tomography (CT scan) of weaned off CPB easily without any inotropes. A left pleu-the chest may be helpful to reveal pericardial tamponade. ral and mediastinal drain was inserted. Following closure of the chest, he was transferred to the intensive care unit (ICU), where he made excellent progress initially and wasCase presentationA 73 year old, hypertensive and hypercholestremic gentle- extubated within 12 hours. At 24 hours post-operatively,man, presented to the Emergency Department with acute the blood pressure (BP) was 110/85 mm Hg, the cardiac index (CI) was 3.0 litres/min/m2 and the total amount ofonset of severe retrosternal chest pain. He had no othersignificant co-morbidities. ECG showed ST segment blood in the drains was 1350 mls, with no drainage in thedepression in leads I, AVL, V5 and V6. The troponin I level last 2 hours. Within 2 hours of removing the drains, thewas 4.1 ng/ml. A diagnosis of non-ST elevation myocar- BP dropped to 80/40 mmHg with a CI of 1.8 litres/min/ m2 with no change in the central venous pressure (CVP,dial infarction (NSTEMI) was made. The patient was givenaspirin, clopidogrel and subcutaneous clexane. During 10 mm Hg), whilst the urine output was maintained atthe admission he continued to get chest pain intermit- >0.5 ml/kg/hr. The systemic vascular resistance was 1150 dynes/cm5. No new changes were seen in the ECG.tently, which required intravenous glyceryl trinitrate infu-sion. A coronary angiogram was performed 4 days later,which revealed significant stenosis of the proximal left A TTE was performed by an experienced sonographeranterior descending artery (LAD) and circumflex artery which showed similar left ventricular function as before(Cx) as well as an occluded right coronary artery (RCA) in and no evidence of pericardial collection or tamponade.the mid-vessel. A TTE showed moderately impaired left In view of depressed LV function, 0.05 mcg/kg/min ofventricular ejection fraction (Journal of Medical Case Reports 2007, 1:162 http://www.jmedicalcasereports.com/content/1/1/162and the urine output was 30 mls/hr. Despite a normal namic instability while in the remaining one-third, collec-TTE, a strong suspicion of pericardial tamponade was tions around the right atrium and/or right ventricle are themade. A trans-oesophageal echocardiogram (TOE) was cause [3]. The decision to re-explore the chest should benot available and it was decided to perform a CT scan of based on clinical suspicion derived from signs whichthe ch ...
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